https://www.selleckchem.com/pr....oducts/salinomycin.h
We further show that genetic disruption of CALML4 within enterocytes results in brush border assembly defects that mirror the loss of other IMAC components, and that CALML4 can also associate with the Usher complex component myosin-7a. Our study further defines the molecular composition and protein-protein interaction network of the IMAC and Usher complex, and may also shed light on the etiology of the sensory disorder USH1H. Published under license by The American Society for Biochemistry and Molecular Biology, Inc.Assembled a-synu
Aimer
Commentaire
Partagez
