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Proinflammatory cytokines were additionally altered in γδ T cell-deficient mice, with additional production of interleukin-1β, interleukin-6 and tumor necrosis aspect. γδ T cells did not seem to add significantly towards the creation of IL-17A or the chemokines CXCL1 and CXCL2. Notably, number survival could be improved by suppressing tumour necrosis factor signaling utilizing the soluble receptor construct etanercept. These findings demonstrate that γδ T cells play a protective par