https://www.selleckchem.com/products/ipa-3.html
05) and obviously improved renal tissue damages induced by triptolide including cell swelling, vacuolization and spotty necrosis. Arctiin significantly inhibited triptolide-induced cytotoxicity in HK-2 cells and increased the cell viability at 24 h ( 0.05). Arctiin also attenuated triptolide-induced cell morphological changes, decreased cell apoptosis rate ( 0.05) and reversed the expressions of IκBα and nuclear p65 ( 0.05). Arctiin can protect the kidney from triptolide-induced damages in rats possibly through the anti-inf
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