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Together, this study reveals specific synaptic and morphological deficits caused by SHANK3 hemizygosity in human cortical neurons at different developmental stages under physiological conditions and validates the use of co-engrafted control and mutant human neurons as a new platform for studying connectivity deficits in genetic neurodevelopmental disorders associated with autism.Reduced gut-microbial diversity ("gut dysbiosis" has been associated with an anhedonic/amotivational syndrome ("sickness behavior" that manifests across seve
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