https://www.selleckchem.com/pr....oducts/hc-030031.htm
In conclusion, this study indicated that through the sulfhydrylation of Keap1, GYY4137 activated the Nrf2/ARE pathway and exerted anti-inflammatory, anti-apoptotic and antioxidant effects in septic mice that consequently protected the integrity of the BBB and improved the clinical outcome of sepsis. Our findings suggest that GYY4137 might be a promising agent for the treatment of SAE. We aimed to investigate predictors of scaffold failure and the potential impact of an optimized scaffold implantation technique by means of a learning c
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