https://bezafibrateagonist.com..../selective-slight-co
In an in vitro style of podocytopathy elicited by a diabetic milieu, triptolide, the major active component of TwHF, at reasonable doses, potentiated the useful effectation of cyclosporine A, and protected podocytes against diabetic milieu-elicited injury, mitigated cytoskeleton derangement, and preserved podocyte filtration barrier function, entailing a synergistic cytoskeleton-preserving and podocyte protective aftereffect of triptolide and cyclosporine A. Mechanistically, inhibitory phos
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