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© The Author(s) 2020.Patients with rheumatoid arthritis (RA) develop features of accelerated ageing, including immunosenescence. These modifications include diminished thymic functionality, development of late-differentiated effector T cells, enhanced telomeric attrition, and extortionate production of cytokines (senescence-associated secretory phenotype). The progression of RA has been linked to the early development of age-related co-morbidities, including weakening of bones, cardiovascular comp