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These actions tend to be mediated by GLP1-R and separate from sperm-secreted insulin. The publicity to Ex-4 fueled the PI3K/AKT signaling and corrected by H89, indicating a PKA-dependence of GLP1/GLP1-R signaling. It appeared that in sperm, insulin secretion regulated by Ex-4 would not occur in a strictly glucose-dependent fashion. A stimulatory activity of Ex-4/GLP1-R on LDH and G6PDH tasks ended up being seen. The Ex-4/GLP1-R reduced triglycerides content concomitantly to an advanced lipase a
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