https://www.selleckchem.com/products/sgi-1027.html
containing the C. albicans skin infection in vivo. CONCLUSION TRAF1-deficient mice can better control fungal infection in the skin, a process attributable to the CXCL-neutrophil axis. Mechanistically, TRAF1 likely regulates CXCL1 expression in both macrophages and keratinocytes through the transcriptional factor NFκB and STAT1, respectively. Our finding offers new insight into the understanding of the immune regulatory mechanisms in host defense against C. albicans infection.BACKGROUND Lysosomal acid lipase deficiency (LALD) is an auto
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