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Results In vitro cellular viability assay and immunoblotting demonstrated that PTEN reduction had been dramatically correlated with AZD8186 sensitivity in triple bad breast cancer (TNBC) cell outlines. Colony formation assay confirmed susceptibility of PTEN-deficient cellular outlines to AZD8186. AZD8186 inhibited PI3K signaling in PTEN loss TNBC cells. AZD8186 in conjunction with paclitaxel, eribulin had synergistic effects on