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Further, hCG treatment after JNK inhibition failed to correct the luteal defect and promote P4 output. Similar to hCG, LH treatment improved luteal function as well and this action of LH was associated with JNK activation in the luteal GCs. These findings could be important from the perspective of corpus luteum biology and luteal phase in human because we for the first time identify a critical role for JNK signaling pathway downstream LH receptor activation by hCG/LH in luteal granulosa cells. © The Author(s) 2020. Published by Oxford University Press on be
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