https://www.selleckchem.com/products/vh298.html
Furthermore, miR‑374a‑5p directly regulated NFAT5. Interestingly, miR‑374a‑5p also contributed to the transformation of microglia with OGD/R treatment from M1 to M2 and suppressed relative expression levels of inflammatory factors by inhibiting NFAT5 in CHME5 cells. Knockdown of NEAT1 regulated OGD/R injury of CHME5 cells via miR‑374a‑5p/NFAT5 axis to induce the shifting of microglia from M1 to M2 and inhibit inflammatory response, making it a potential target for stroke treatment.Morphine and tramadol are the opioid analgesic drugs actin
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