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Consequently we have heard of introduction of biologics which either target T-cell receptors or restrict Tumour Necrosis Factor-alpha (TNF-α) or prevent interleukins (IL) like IL-12, IL-17, IL-17 receptor and more recently IL-23. Medications particularly focusing on the p19 subunit of IL-23 have shown promising leads to the management of chronic plaque psoriasis. It has offered solution to the development of a unique course of biologics, that is, the IL-23p19 inhibitors having a significantly better safety profile as compared to
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