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In addition, the treatment with exogeneous tryptase in MC-deficient mice can induce the damage observed in wild type mice in the context of liver IR. In vitro, neutrophil infiltration and inflammatory factor secretion were regulated by Tryptase/PAR-2, involving the adhesion molecule expression to regulate neutrophil adhesion dependent on NF-κB pathway. Conclusion tryptase/PAR-2 participates in liver injury through the activation of LSECs in the early phase of liver IR.The complement system is an essential component of innate immunity. I
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