https://www.selleckchem.com/pr....oducts/l-nmma-acetat
The general anesthetic etomidate, which acts through GABAA receptors, impairs the formation of new memories under anesthesia. This study addresses the molecular and cellular mechanisms by which this occurs. Here, using a new line of genetically engineered mice carrying the GABAAR β2-N265M mutation, we tested the roles of receptors that incorporate GABAA receptor β2 vs. β3 subunits to suppression of long-term potentiation (LTP), a cellular model of learning and memory. We found that brain slices from β2-N265M mice resisted etomida
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