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Additionally, the growth inhibitory aftereffect of chemical 10 on MDA-MB-231 cells ended up being involving cellular cycle arrest and apoptosis. Norhierridin B (1 interfered with a few p53 transcriptional targets, increasing p21, Bax, and MDM2, while reducing Bcl-2 necessary protein amounts, which recommended the potential activation of a p53 pathway. Entirely, these results evidenced a good enhancement associated with the antitumor activity of hydroquinone 10 compared to 6 and i
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