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This method ended up being regulated by nucleocytoskeletal connections, Par3 clustering proximal to nuclear lamina folds, and retrograde motion of actin bundles that correlated with nuclear ruptures. Considerably, Par3 additionally regulated the occurrence of natural nuclear ruptures facilitated because of the downmodulation of lamins. This work uncovered a novel role for Par3 in controlling the actin-dependent causes functioning on the nuclear envelope to remode
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