https://www.selleckchem.com/pr....oducts/folinic-acid.
In contrast, the IL-18BP was decreased in the lamina propria in asthma and was absent in the bronchial epithelium. IL-18 was released in sputum with IL-18BP elevated in patients with asthma. The IL-18Rα expression was not different between health and disease. IL-18-stimulated bronchial epithelial cells increased intracellular calcium, wound repair, metabolic activity, morphological changes and epithelial cellular differentiation. In asthma, the dynamic interaction between IL-18, its cognate receptor and natural inhibitor is complex
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