https://www.selleckchem.com/products/gdc-0994.html
Notably, knockdown of Cdk5 significantly abrogated the effects of on cellular processes and Wnt/β-catenin signaling in relation to the progression of CRC. The results of this study demonstrate that orchestrates a molecular network involving the direct role of Cdk5 in activating Wnt/β-catenin signaling to modulate CRC progression. Thus, in-depth investigations of associated molecular pathways may offer valuable insight into the pathogenesis of CRC, which may help further the development of treatment for this disease. The results of this
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