https://www.selleckchem.com/pr....oducts/sodium-2-1h-i
The levels of microRNA-155, VCAM1 and ICAM1 were increased in sEV-treated hCMEC/D3 cells. Blocking the expression of VCAM1, but not of ICAM1, prevented sEV-mediated T-cell adhesion to brain endothelia. These results suggest that sEVs derived from inflamed BECs promote cerebrovascular dysfunction. These findings may provide new insights into the mechanisms involving neuroinflammatory disorders.Three-dimensional (3D) printing is among the rapidly evolving technologies with applications in many sectors. The pharmaceut
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