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Furthermore, auranofin paid down the burden of intracellular N. gonorrhoeae by over 99% outperforming the medication of choice ceftriaxone. Auranofin had been discovered more advanced than ceftriaxone in reducing the release of the pro-inflammatory cytokine IL-8 by endocervical cells contaminated with N. gonorrhoeae. Additionally, auranofin exhibited an extended post-antibiotic impact over 10 h, also failure to create resistant mutants. Overall, the existing
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