https://www.selleckchem.com/mTOR.html
The consequence is the formation of one or more pathogenic proteins, some of which may exhibit 'prion-like' behaviour and propagate through the brain from initial sites of formation along neuro-anatomical pathways to affect connected brain regions. Variations in the pathological proteins formed and their anatomical spread are ultimately responsible for the clinical and patholo-gical diversity of disease phenotypes. Minimising the factors which contribute to the allostatic load over a lifetime and maximising cognitive and physical exercise may be ne
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